Cataracts and Eyelammation

Correlation Between Cataracts and Eyelammation

Unlike the other body parts, like the ribs, stomach, muscles, bones, or other connective tissues, the interior portion of the shark’s eye lens does not continuously develop as the animal grows older. It begins to shrivel up when it is about twenty years old and gradually turns into a thin crusty layer which makes the eye lens less visible. This thin crusty layer makes it more susceptible to damage and abrasion. Eye lens can be damaged easily because of the frequent rubbing against objects like rocks, metal objects, sand, water, and other objects. Some other physical trauma like being hit by a light ray also can cause damage. Stress, strain, improper care of the eye lens and injuries may also cause this crusty layer to grow faster and make it harder for the eye lens to function properly.


Like other body parts

the structure of the shark’s eye requires a regular and adequate supply of fluids to ensure that it remains in good health and active. However, for some species, especially those that live in the water where oxygen is scarce like the Greenland shark, eye fluid is absent and the only way for the shark’s eye to function is through its retina, which consists mainly of crystalline lens cells. In these cases, the shark must absorb visual information through its right pair of eyes, called the sense organs, which are located in the top portion of its head.


To have a clear and perfectly functional eye lens

a healthy retina needs to be able to absorb visual information properly and process it properly. This is where the work of the Bloome is important. The Bloome is a collection of lens cells (two per eye) that lie within the layers of the retinal pigment epithelial layer. These bloatome cells are responsible for the regulation and production of new vision cells. Bloemendal H has a broad spectrum of activity, which is needed for the proper generation of new cells and the formation of new connections between existing cells.


Bloemendal H

is a unique protein complex, which belongs to the class of structural proteins called cysteine-rich amyloid. Cysteine-rich amyloids are insoluble fibrous protein aggregates that form clumps within the eye lens surface. Eye inflammation and damage, caused by certain environmental factors including exposure to certain toxins, can result in the accumulation of these amyloid clumps, which become crystallized and forms small painful balls (called ocular balls) which can irritate the retina and can ultimately lead to cataract and glaucoma. In the case of humans, this can result in the development of puffy, red eyes, commonly known as hazy or blue eyes, with associated pain and blurred vision. When this condition is caused by the accumulation of excess proteins, called hyaluronic acid, it can cause painful ulcers and the development of the condition known as a hematoma.


Truscott-Hicks (Trouse et al., 2021)

is an interesting study that investigated whether there is a relationship between inflammatory eye disease and the metabolic disorder Truscott-Hicks (Trouse, unpublished data), which affects around one-quarter of all adults. The symptoms of Touse-Hicks consist of red eyes, dry skin, irritation, itching, and a scratchy feeling. The researchers looked at two groups of patients, one who had cataracts and one without, and found that those with cataracts had higher levels of TSH (trial analysis confirmed). In addition, the researchers found that there was a significant positive association between metabolic disorder and TSH, supporting the hypothesis that TSH may be an important link between eye inflammation and cataract. This was an important study, because TSH has the ability to stimulate inflammatory responses in the body, which may affect the development of cataracts, and may also be a risk factor for the development of various eye diseases including cataracts, macular degeneration, and vitreous choroid. However, more studies will have to confirm these results, and they are currently being conducted to examine whether there are other risk factors associated with the metabolic syndrome that are not associated with eye inflammation.


As far as I’m concerned the most important finding from this article

which is that there is a strong link between eye inflammation and metabolic syndrome, is that those people with the syndrome have higher levels of crystalline proteins in their urine, which is a strong indicator of structural stress throughout the body and strongly suggests the possibility of eye damage. If this is true, then the correlation between cataracts and the development of eye inflammation is likely to hold. A cataract is a well-known condition, and there are many cataract surgery treatments available for those with mild to severe forms of the disease. However, any link between the two should still be explored further.

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